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doi: 10.3892/mmr.2023.12932 138 MeM

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In general, iNOS-NO observed in Hp infected gastric mucosa and particularly in the Hp-originating GC supports the proposal of Correa (110, 111) that Hp induced inflammatory changes in gastric mucosa results from the action of numerous ROS and RNS and, therefore, the carcinogenesis could be considered as a result of oxidative stress ( Table 4 )

These cells are essential for preserving lung homeostasis because they have the ability to develop into a variety of lung cell types, such as endothelial and alveolar epithelial cells (86)