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It may reach the correct site, but may not be targeted toward the specific agent responsible for oxidative damage, as observed in the case of vitamin E that inhibits lipid peroxidation, but may not prevent the increased oxidative DNA, RNA, and protein damage seen in Alzheimers disease (Pratic, 2008)

Hydrogen peroxide can be formed by a two-electron reduction of dioxygen (O 2 + 2e O 2 2 ) or a one-electron reduction of superoxide radical anion (O 2 + e O 2 2 ) by superoxide dismutases (see below) (Jomova et al