modelled a ROS-dependent mitochondrial oscillator, which included modelling the kinetics of glutathione peroxidase and reductase (Cortassa et al., 2004)
Jiang K, Cao X, Wu H, Xu Y, Liu L, Qian H, et al
The mechanisms of TB-500 are still being investigated, but known actions of TB4 include: Binding as an actin-binding protein to inhibit the polymerization of globular actin (G-actin) into filamentous actin (F-actin), known as actin sequestration, which results in higher G-actin levels and increased cellular motility [23, 24] TB4s ability to increase cellular motility may help progenitor cells to reach sites of injury and initiate tissue recovery [25]
[DOI] [PubMed] [Google Scholar] 89.Suh J.H., Zhu B.Z., de Szoeke E., Frei B., Hagen T.M