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In malignant tumors, IFN produced by CD8+ T cells increases with rising Gln concentrations, indicating that immunotherapy targeting tumors also relies on Gln-dependent activation of CD8+ T cells ( In the microenvironment of liver cancer tumors, Gln deficiency promotes the invasion of myeloid cells expressing the immunosuppressive receptor GPR109A, facilitating immune evasion

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The pathogenic variant of TM6SF2 increases the risk and severity of NASH but reduces cardiovascular disease, seemingly by reducing triglyceride secretion from the liver as VLDL (117)

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