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In parallel, studies have reported modulation of early growth response-1 (EGR-1) expression and its regulatory partner NAB2, which are involved in cytokine signaling and early extracellular matrix formation during cellular response processes [2]

For example, sustained stimulation by inflammatory cytokines such as IL-1 and TNF-, persistent oxidative stress, and pathological iron overload states have all been shown to significantly suppress SLC7A11 expression or its transport activity (Xiao et al., 2024